Auto Afghani
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Terpenes
Auto Afghani effects are mostly calming.
Auto Afghani
Auto Afghani is a modern, auto-flowering variety resulting from a cross between Skunk No. 1 and Ruderalis. The specific breeding history of this strain remains undocumented, as no official record identifies its origin or the intent behind its initial development. Its growth cycle is defined by a 60-day flowering period, and it is recognized for an easy cultivation difficulty. Suitable for both indoor and outdoor environments, the strain is known to produce a low yield.
The terpene profile of Auto Afghani is dominated by myrcene, followed by caryophyllene, limonene, pinene, terpinolene, linalool, beta-pinene, humulene, nerolidol, ocimene, bisabolol, guaiol, camphene, caryophyllene-oxide, and p-cymene. These compounds contribute to the strain’s sensory profile, with an aroma and flavor that likely reflect the interplay of these fifteen confirmed terpenes. The sequence of the profile follows the recorded dominance, descending from the primary myrcene content through the trace amounts of p-cymene.
As a THC-dominant cultivar, Auto Afghani is reported to induce effects that are relaxed, happy, and uplifted. These characteristics make it a functional choice for users seeking relief from stress and pain, or support for sleep. While it serves as a modern genetic variety, there are no recorded notable offspring currently associated with this strain in contemporary breeding programs.
Terpene Profile
Synergies (+) and conflicts (−) are relative to each other within this profile.
| Terpene | Share | Character | Likely role |
|---|---|---|---|
| myrcene | ~60% | earthy | relaxing · solo |
| caryophyllene | ~28% | spicy | relaxing · social |
| limonene | ~12% | citrus | social · creative |
Research notes below describe isolated terpene mechanisms and early findings. They do not guarantee effects from this strain and are not medical advice.
Russo 2011: naloxone-sensitive analgesia, potentiates barbiturate sleep; dominant sedating terpenoid; blocks hepatic carcinogenesis by aflatoxin.
~28%
spicy
●●○○
Russo 2011: only terpene that is a selective full CB2 agonist (100 nM); Gertsch et al. 2008: acts as dietary cannabinoid; unique anti-inflammatory and gastric cytoprotective properties.
Russo 2011: increases serotonin in prefrontal cortex + dopamine in hippocampus via 5-HT1A; Johns Hopkins 2024: significantly reduced anxiety vs THC alone.
Effects
Reported effects — derived from terpene chemistry and cannabinoid profile.
relaxed
eveningPrimary endpoint of myrcene+linalool sedating combinations; GABA modulation is the dominant mechanistic driver.
happy
anytimeuplifted
morningLimonene anxiolytic/antidepressant via serotonin elevation in prefrontal cortex (Russo 2011); mood improvement without full euphoria; key for balanced-1-1 profiles.
Genetic Profile
Autoflower
Photoperiod-independent. Flowers based on age, not light cycle. Compact and fast.
THC-Dominant
High THC, trace CBD. Psychoactive. Full CB1 agonism — euphoria, appetite, analgesia.
Genealogy
Parentage, ancestry, and genetic relatives of Auto Afghani.
Ancestry
Great-great-grandparents
Great-grandparents
Grandparents
Siblings
Share parents skunk no 1 / ruderalis
Composite Traits
Where to buy Auto Afghani
No verified dispensaries on file — find it on Leafly.
Dispensary Locator
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What would Auto Afghani × ? produce?
Predict the terpene profile, effects, and growing traits of a cross. Our gene weaver engine votes on dominant traits from both parents.
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Frequently Asked Questions
Is Auto Afghani indica or sativa?
Auto Afghani is modeled here as a autoflower (photoperiod-independent).
What terpene is dominant in Auto Afghani?
Myrcene is shown as the dominant terpene at approximately ~60%. Caryophyllene follows as the secondary terpene.
Is Auto Afghani good for daytime use?
Auto Afghani is versatile and works across different times of day depending on dose and individual response.
How accurate is this data?
See the "Data confidence" card in the sidebar. Terpene profiles and effects are chemistry-informed estimates — individual responses depend on phenotype, source, and personal chemistry.