APNMauto
AutoflowerModernTHC-Dominantevening

Auto Pain no More

Top flavors

earthycitrusspicy
CalmingEnergizing

Auto Pain no More effects are mostly calming.

Auto Pain no More is a modern, auto-flowering cannabis strain developed through the hybridization of Bubblegum and Ruderalis. Due to the limited documentation surrounding its development, the specific origins and breeders responsible for this genetic pairing remain unrecorded. As an auto-flowering variety, it is designed for ease of growth in both indoor and outdoor environments, completing its flowering stage within 70 days. While it provides a reliable growth cycle for varied cultivators, the strain is known for producing a low yield.

The chemical character of Auto Pain no More is defined by a complex terpene profile that prioritizes myrcene, followed by limonene, caryophyllene, and linalool. The secondary and tertiary tiers consist of pinene, beta-pinene, terpinolene, humulene, ocimene, nerolidol, camphene, bisabolol, and guaiol. This specific sequence of aromatic compounds dictates the strain's sensory expression, offering a nuanced bouquet that balances earthy and citrus notes with subtle spicy and floral undertones.

This THC-dominant strain is primarily utilized for its therapeutic potential, serving as an effective tool for the management of stress, physical pain, and sleep difficulties. Users typically report feelings of relaxation, happiness, and an uplifted state of mind following consumption. To date, there are no recorded notable offspring descending from this variety, leaving Auto Pain no More to be primarily regarded for its individual effects rather than its utility in future breeding programs.

Terpene Profile

Synergies (+) and conflicts (−) are relative to each other within this profile.

myrcene ~60%limonene ~28%caryophyllene ~12%
TerpeneShare
myrcene~60%
limonene~28%
caryophyllene~12%

Research notes below describe isolated terpene mechanisms and early findings. They do not guarantee effects from this strain and are not medical advice.

~60%

earthy

●●●○

Russo 2011: naloxone-sensitive analgesia, potentiates barbiturate sleep; dominant sedating terpenoid; blocks hepatic carcinogenesis by aflatoxin.

~28%

citrus

●●○○

Russo 2011: increases serotonin in prefrontal cortex + dopamine in hippocampus via 5-HT1A; Johns Hopkins 2024: significantly reduced anxiety vs THC alone.

~12%

spicy

●●○○

Russo 2011: only terpene that is a selective full CB2 agonist (100 nM); Gertsch et al. 2008: acts as dietary cannabinoid; unique anti-inflammatory and gastric cytoprotective properties.

Effects

Reported effects — derived from terpene chemistry and cannabinoid profile.

Genetic Profile

Autoflower

Photoperiod-independent. Flowers based on age, not light cycle. Compact and fast.

THC-Dominant

High THC, trace CBD. Psychoactive. Full CB1 agonism — euphoria, appetite, analgesia.

Genealogy

Parentage, ancestry, and genetic relatives of Auto Pain no More.

Ancestry

Auto Pain no MoreFull tree →
View full lineage tree →

Composite Traits

Use caution if

daytime-productivitymorning-useevening-wind-down

Where to buy Auto Pain no More

No verified dispensaries on file — find it on Leafly.

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What would Auto Pain no More × ? produce?

Predict the terpene profile, effects, and growing traits of a cross. Our gene weaver engine votes on dominant traits from both parents.

Build a cross with Auto Pain no More

Similar strains

Same primary terpene with overlapping effects.

Frequently Asked Questions

Is Auto Pain no More indica or sativa?

Auto Pain no More is modeled here as a autoflower (photoperiod-independent).

What terpene is dominant in Auto Pain no More?

Myrcene is shown as the dominant terpene at approximately ~60%. Limonene follows as the secondary terpene.

Is Auto Pain no More good for daytime use?

Auto Pain no More is versatile and works across different times of day depending on dose and individual response.

How accurate is this data?

See the "Data confidence" card in the sidebar. Terpene profiles and effects are chemistry-informed estimates — individual responses depend on phenotype, source, and personal chemistry.