Bounty Killer
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Terpenes
Bounty Killer has balanced effects.
Bounty Killer
Bounty Killer is a modern hybrid-sativa cultivar bred from the crossing of Chemdog and DeadHead OG. Information regarding the original breeder and the specific timeline of its development remains undocumented, reflecting the common ambiguity surrounding the origins of many contemporary hybrid varieties. As a product of these genetics, it is grown in both indoor and outdoor environments, demonstrating a moderate cultivation difficulty and a high yield potential over a 70-day flowering cycle.
The sensory profile of this strain is defined by a complex terpene hierarchy, with caryophyllene serving as the dominant terpene, followed by myrcene, limonene, linalool, humulene, bisabolol, beta-pinene, terpinolene, and pinene. Users can expect a nuanced aromatic experience consistent with this specific array of compounds. The high concentration of caryophyllene and myrcene provides a distinct foundation for its scent and flavor, while the secondary presence of limonene and linalool adds significant layers to the overall profile.
THC-dominant in its cannabinoid composition, Bounty Killer is characterized by its creative, uplifted, and happy effects. These qualities make it a suitable choice for tasks requiring creativity, social engagement, and focus. To date, there are no recorded instances of notable offspring attributed to this strain, leaving it as a singular entry in the contemporary horticultural landscape.
Terpene Profile
Synergies (+) and conflicts (−) are relative to each other within this profile.
| Terpene | Share | Character | Likely role |
|---|---|---|---|
| caryophyllene | ~60% | spicy | relaxing · social |
| myrcene | ~28% | earthy | relaxing · solo |
| limonene | ~12% | citrus | social · creative |
Research notes below describe isolated terpene mechanisms and early findings. They do not guarantee effects from this strain and are not medical advice.
~60%
spicy
●●○○
Russo 2011: only terpene that is a selective full CB2 agonist (100 nM); Gertsch et al. 2008: acts as dietary cannabinoid; unique anti-inflammatory and gastric cytoprotective properties.
Russo 2011: naloxone-sensitive analgesia, potentiates barbiturate sleep; dominant sedating terpenoid; blocks hepatic carcinogenesis by aflatoxin.
Russo 2011: increases serotonin in prefrontal cortex + dopamine in hippocampus via 5-HT1A; Johns Hopkins 2024: significantly reduced anxiety vs THC alone.
Effects
Reported effects — derived from terpene chemistry and cannabinoid profile.
creative
morningAssociated with terpinolene/limonene profiles; dopaminergic and serotonergic modulation via limonene 5-HT1A supports divergent thinking.
uplifted
morningLimonene anxiolytic/antidepressant via serotonin elevation in prefrontal cortex (Russo 2011); mood improvement without full euphoria; key for balanced-1-1 profiles.
happy
anytimeGenetic Profile
Sativa-dominant
Primarily sativa with indica grounding. Uplifting with body relaxation.
THC-Dominant
High THC, trace CBD. Psychoactive. Full CB1 agonism — euphoria, appetite, analgesia.
Genealogy
Parentage, ancestry, and genetic relatives of Bounty Killer.
Ancestry
Great-great-grandparents
Great-grandparents
Grandparents
Siblings
Share parents chemdog / deadhead og
Composite Traits
Where to buy Bounty Killer
No verified dispensaries on file — find it on Leafly.
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What would Bounty Killer × ? produce?
Predict the terpene profile, effects, and growing traits of a cross. Our gene weaver engine votes on dominant traits from both parents.
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Frequently Asked Questions
Is Bounty Killer indica or sativa?
Bounty Killer is modeled here as a sativa-dominant (primarily sativa with indica grounding).
What terpene is dominant in Bounty Killer?
Caryophyllene is shown as the dominant terpene at approximately ~60%. Myrcene follows as the secondary terpene.
Is Bounty Killer good for daytime use?
Bounty Killer is versatile and works across different times of day depending on dose and individual response.
How accurate is this data?
See the "Data confidence" card in the sidebar. Terpene profiles and effects are chemistry-informed estimates — individual responses depend on phenotype, source, and personal chemistry.