Death by Lemons
20.9%
THC
Top flavors
Terpenes
Death by Lemons has balanced effects.
Death by Lemons potency is average.
Death by Lemons
20.9%
THC
Top flavors
Terpenes
Death by Lemons has balanced effects.
Death by Lemons potency is average.
Death by Lemons is a modern hybrid-sativa cultivar derived from a Lemon G lineage. Developed for those seeking a specific balance of sativa-leaning traits, it performs well in both indoor and outdoor environments. Growers can expect a moderate cultivation difficulty and a high yield, provided they are prepared for a 66-day flowering cycle.
The aromatic and flavor profile is defined by a complex chemical composition, led by caryophyllene and supported by nerolidol and myrcene. This foundation is further layered with notes of limonene, humulene, linalool, beta-pinene, pinene, camphene, geraniol, ocimene, and terpinolene. This specific arrangement of terpenes creates a distinct sensory experience that reflects its clear Lemon G roots.
With a THC concentration of approximately 20.9 percent, Death by Lemons serves as a reliable option for users prioritizing creative, uplifted, and happy effects. It is particularly well-suited for social engagements, tasks requiring focused attention, or the pursuit of creative endeavors. To date, there are no recorded offspring attributed to this specific genetic profile.
Terpene Profile
Synergies (+) and conflicts (−) are relative to each other within this profile.
| Terpene | Share | Character | Likely role |
|---|---|---|---|
| caryophyllene | ~60% | spicy | relaxing · social |
| nerolidol | ~28% | floral | sleep · relaxing |
| myrcene | ~12% | earthy | relaxing · solo |
Research notes below describe isolated terpene mechanisms and early findings. They do not guarantee effects from this strain and are not medical advice.
~60%
spicy
●●○○
Russo 2011: only terpene that is a selective full CB2 agonist (100 nM); Gertsch et al. 2008: acts as dietary cannabinoid; unique anti-inflammatory and gastric cytoprotective properties.
Russo 2011: sedative properties; enhances transdermal pharmaceutical penetration; antimalarial; PMC11060501: GABAergic-mediated antinociception.
Russo 2011: naloxone-sensitive analgesia, potentiates barbiturate sleep; dominant sedating terpenoid; blocks hepatic carcinogenesis by aflatoxin.
Effects
Reported effects — derived from terpene chemistry and cannabinoid profile.
creative
morningAssociated with terpinolene/limonene profiles; dopaminergic and serotonergic modulation via limonene 5-HT1A supports divergent thinking.
uplifted
morningLimonene anxiolytic/antidepressant via serotonin elevation in prefrontal cortex (Russo 2011); mood improvement without full euphoria; key for balanced-1-1 profiles.
happy
anytimeGenetic Profile
Sativa-dominant
Primarily sativa with indica grounding. Uplifting with body relaxation.
THC-Dominant
High THC, trace CBD. Psychoactive. Full CB1 agonism — euphoria, appetite, analgesia.
Genealogy
Parentage, ancestry, and genetic relatives of Death by Lemons.
Ancestry
Great-great-grandparents
Great-grandparents
Grandparents
Siblings
Share parent lemon g
Composite Traits
Where to buy Death by Lemons
No verified dispensaries on file — find it on Leafly.
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What would Death by Lemons × ? produce?
Predict the terpene profile, effects, and growing traits of a cross. Our gene weaver engine votes on dominant traits from both parents.
Build a cross with Death by Lemons →Similar strains
Same primary terpene with overlapping effects.
Frequently Asked Questions
Is Death by Lemons indica or sativa?
Death by Lemons is modeled here as a sativa-dominant (primarily sativa with indica grounding).
What terpene is dominant in Death by Lemons?
Caryophyllene is shown as the dominant terpene at approximately ~60%. Nerolidol follows as the secondary terpene.
Is Death by Lemons good for daytime use?
Death by Lemons is versatile and works across different times of day depending on dose and individual response.
How accurate is this data?
See the "Data confidence" card in the sidebar. Terpene profiles and effects are chemistry-informed estimates — individual responses depend on phenotype, source, and personal chemistry.