Killer A5 Haze
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Terpenes
Killer A5 Haze effects are mostly calming.
Killer A5 Haze
Killer A5 Haze is a modern hybrid-sativa derived from a cross between Original Haze and an Africa Sativa. The specific historical circumstances surrounding its precise origin or the individual breeder responsible remain undocumented. This strain is cultivated in both indoor and outdoor environments, demonstrating a moderate difficulty level and requiring a 74-day flowering period to reach a high yield.
The chemical expression of this strain is defined by a distinct terpene profile, which is led by nerolidol, followed by limonene, caryophyllene, humulene, linalool, myrcene, bisabolol, beta-pinene, caryophyllene-oxide, camphene, terpinolene, and pinene. As a THC-dominant cultivar, its aromatic complexity originates from this specific arrangement of compounds. The sensory experience is dictated by this confirmed hierarchy of terpenes, contributing to the strain's nuanced aromatic profile.
Users seeking a cerebral experience often turn to this strain for its capacity to bolster creativity, social interaction, and focus. The effects are consistently categorized as creative, uplifted, and happy. Because it is a modern hybrid, there are no notable offspring currently recorded in the lineage. Its utility remains centered on these specific cognitive and mood-enhancing applications, supported by its balanced sativa-leaning genetic background.
Terpene Profile
Synergies (+) and conflicts (−) are relative to each other within this profile.
| Terpene | Share | Character | Likely role |
|---|---|---|---|
| nerolidol | ~60% | floral | sleep · relaxing |
| limonene | ~28% | citrus | social · creative |
| caryophyllene | ~12% | spicy | relaxing · social |
Research notes below describe isolated terpene mechanisms and early findings. They do not guarantee effects from this strain and are not medical advice.
Russo 2011: sedative properties; enhances transdermal pharmaceutical penetration; antimalarial; PMC11060501: GABAergic-mediated antinociception.
Russo 2011: increases serotonin in prefrontal cortex + dopamine in hippocampus via 5-HT1A; Johns Hopkins 2024: significantly reduced anxiety vs THC alone.
~12%
spicy
●●○○
Russo 2011: only terpene that is a selective full CB2 agonist (100 nM); Gertsch et al. 2008: acts as dietary cannabinoid; unique anti-inflammatory and gastric cytoprotective properties.
Effects
Reported effects — derived from terpene chemistry and cannabinoid profile.
creative
morningAssociated with terpinolene/limonene profiles; dopaminergic and serotonergic modulation via limonene 5-HT1A supports divergent thinking.
uplifted
morningLimonene anxiolytic/antidepressant via serotonin elevation in prefrontal cortex (Russo 2011); mood improvement without full euphoria; key for balanced-1-1 profiles.
happy
anytimeGenetic Profile
Sativa-dominant
Primarily sativa with indica grounding. Uplifting with body relaxation.
THC-Dominant
High THC, trace CBD. Psychoactive. Full CB1 agonism — euphoria, appetite, analgesia.
Genealogy
Parentage, ancestry, and genetic relatives of Killer A5 Haze.
Ancestry
Great-great-grandparents
Grandparents
Siblings
Share parents original haze / africa sativa
Composite Traits
Where to buy Killer A5 Haze
No verified dispensaries on file — find it on Leafly.
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What would Killer A5 Haze × ? produce?
Predict the terpene profile, effects, and growing traits of a cross. Our gene weaver engine votes on dominant traits from both parents.
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Frequently Asked Questions
Is Killer A5 Haze indica or sativa?
Killer A5 Haze is modeled here as a sativa-dominant (primarily sativa with indica grounding).
What terpene is dominant in Killer A5 Haze?
Nerolidol is shown as the dominant terpene at approximately ~60%. Limonene follows as the secondary terpene.
Is Killer A5 Haze good for daytime use?
Killer A5 Haze is versatile and works across different times of day depending on dose and individual response.
How accurate is this data?
See the "Data confidence" card in the sidebar. Terpene profiles and effects are chemistry-informed estimates — individual responses depend on phenotype, source, and personal chemistry.