Sour Pinot Autoflowering
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Terpenes
Sour Pinot Autoflowering effects are mostly calming.
Sour Pinot Autoflowering
Sour Pinot Autoflowering is a modern, autoflowering cultivar developed by crossing Grand Daddy Purple with Ruderalis genetics. Classified as a straightforward project for cultivators, this strain is suitable for both indoor and outdoor environments, maintaining a manageable 84-day flowering cycle. While it is known for its accessibility to growers of all experience levels, it generally produces a low harvest yield.
The chemical profile of Sour Pinot Autoflowering is defined by a complex, layered terpene structure beginning with dominant myrcene, followed by caryophyllene and limonene. The secondary tier of the profile includes linalool, terpinolene, and pinene, while the minor aromatic compounds range from beta-pinene, humulene, and ocimene to nerolidol, bisabolol, and guaiol. The remaining trace profile concludes with caryophyllene-oxide and camphene, which collectively shape the specific sensory experience of the strain.
Users of this THC-dominant cultivar typically report effects that are relaxed, happy, and uplifted. These characteristics make the flower a frequent choice for those seeking to address stress, pain, or difficulties with sleep. Beyond its individual use, the strain has expanded its genetic impact through the creation of notable offspring, specifically the cross of Thin Mint with Sour Pinot Autoflower.
Terpene Profile
Synergies (+) and conflicts (−) are relative to each other within this profile.
| Terpene | Share | Character | Likely role |
|---|---|---|---|
| myrcene | ~60% | earthy | relaxing · solo |
| caryophyllene | ~28% | spicy | relaxing · social |
| limonene | ~12% | citrus | social · creative |
Research notes below describe isolated terpene mechanisms and early findings. They do not guarantee effects from this strain and are not medical advice.
Russo 2011: naloxone-sensitive analgesia, potentiates barbiturate sleep; dominant sedating terpenoid; blocks hepatic carcinogenesis by aflatoxin.
~28%
spicy
●●○○
Russo 2011: only terpene that is a selective full CB2 agonist (100 nM); Gertsch et al. 2008: acts as dietary cannabinoid; unique anti-inflammatory and gastric cytoprotective properties.
Russo 2011: increases serotonin in prefrontal cortex + dopamine in hippocampus via 5-HT1A; Johns Hopkins 2024: significantly reduced anxiety vs THC alone.
Effects
Reported effects — derived from terpene chemistry and cannabinoid profile.
relaxed
eveningPrimary endpoint of myrcene+linalool sedating combinations; GABA modulation is the dominant mechanistic driver.
happy
anytimeuplifted
morningLimonene anxiolytic/antidepressant via serotonin elevation in prefrontal cortex (Russo 2011); mood improvement without full euphoria; key for balanced-1-1 profiles.
Genetic Profile
Autoflower
Photoperiod-independent. Flowers based on age, not light cycle. Compact and fast.
THC-Dominant
High THC, trace CBD. Psychoactive. Full CB1 agonism — euphoria, appetite, analgesia.
Genealogy
Parentage, ancestry, and genetic relatives of Sour Pinot Autoflowering.
Ancestry
Great-great-grandparents
Great-grandparents
Grandparents
Siblings
Share parents grand daddy purple / ruderalis
Composite Traits
Where to buy Sour Pinot Autoflowering
No verified dispensaries on file — find it on Leafly.
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What would Sour Pinot Autoflowering × ? produce?
Predict the terpene profile, effects, and growing traits of a cross. Our gene weaver engine votes on dominant traits from both parents.
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Frequently Asked Questions
Is Sour Pinot Autoflowering indica or sativa?
Sour Pinot Autoflowering is modeled here as a autoflower (photoperiod-independent).
What terpene is dominant in Sour Pinot Autoflowering?
Myrcene is shown as the dominant terpene at approximately ~60%. Caryophyllene follows as the secondary terpene.
Is Sour Pinot Autoflowering good for daytime use?
Sour Pinot Autoflowering is versatile and works across different times of day depending on dose and individual response.
How accurate is this data?
See the "Data confidence" card in the sidebar. Terpene profiles and effects are chemistry-informed estimates — individual responses depend on phenotype, source, and personal chemistry.